Music therapy is one of the treatment methods for stroke, which is performed by listening, singing, playing or cooperating with music rhythm. With music stimulation, music therapy can accelerate the nerve repair in patients with stroke, improve the blood flow of brain, the cognitive level and the motor function, and help to alleviate the dysfunction caused by stroke. Compared with the conventional drug therapy, music therapy can also prevent the potential mental disorders and treat the existing mental disorders in patients with stroke, so as to improve the patients’ psychology, effectively supplement the deficiency of psychological intervention, and help the patients to recover better. This paper reviews the latest progress in music therapy for stroke, and looks forward to the future development of music therapy.
Progress in antidepressant and anxiety treatment to improve motor function and its mechanism in poststroke patients
With in-depth research on motor function recovery after stroke, more and more literatures have reported that antidepressant and anxiety treatment is not only effective for post-stroke depression and anxiety, but also significantly promotes the recovery of motor function after stroke. In recent years, it has been studied whether the traditional Chinese and Western medicine drugs against depression and anxiety can improve the recovery of motor function in post-stroke patients, and its mechanism has been discussed. In this paper, the latest progress in the role and mechanism of Chinese and Western medicine in improving or independently improving the limb motor function in post-stroke patients is reviewed, in order to provide references for the prevention, treatment and rehabilitation of limb motor dysfunction after stroke.
Objective: To improve the understanding of mitochondrial encephalomyopathy with lactic acidosis and stroke-like episode (MELAS) syndrome in clinical practice. Methods: The clinical diagnosis and treatment of one patient with MELAS syndrome were reported. The clinical manifestations, diagnosis and possible mechanism of MELAS syndrome were discussed based on literature review. Results: One patient with MELAS syndrome manifested first-onset stroke attack, and then appeared epileptic seizures. Suspicious antibodies in cerebrospinal fluid and serum were negative. The result of mitochondrial disease gene detection revealed m.3243A>G, and the final diagnosis was MELAS syndrome. Conclusion: MELAS syndrome is one of the most common type of mitochondrial myopathy, which can influence multiple systems, including brain and muscle tissues. The clinical manifestations of MELAS syndrome are various and lack of a unified diagnostic standard, so it is easy to escape diagnosis or be misdiagnosed, and the prognosis is poor. Therefore, it is very important to recognize MELAS syndrome early, in order to make the diagnosis as soon as possible and give appropriate treatment to improve the prognosis of patients.
